It's largely about the suppression of early treatment, and the risks of the vaccines.
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My own father, who's vaccinated, got sick with COVID-19 and gave it to me. I'm just getting over it right now. It goofed up my sense of smell and taste, but I didn't have any respiratory issues from it at all. At the first sign of symptoms, I immediately started taking NAC, glycine, quercetin, resveratrol, vitamin D, famotidine, and diphenhydramine. That beat it back pretty solidly.
COVID-19 kills people with low antioxidant capacity by inducing severe oxidative stress.
Multi-system involvement and rapid clinical deterioration are hallmarks of coronavirus disease 2019 (COVID-19) related mortality. The unique clinical phenomena in severe COVID-19 can be perplexing, and they include disproportionately severe hypoxemia ...
www.ncbi.nlm.nih.gov
Although most patients recover from acute COVID-19, some experience postacute sequelae of severe acute respiratory syndrome coronavirus 2 infection (PASC). One subgroup of PASC is a syndrome called “long COVID-19,” reminiscent of myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS)...
www.pnas.org
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is involved in a global outbreak affecting millions of people who manifest a variety of symptoms. Coronavirus disease 2019 (COVID-19) caused by SARS-CoV-2 is increasingly associated with cardiovascular ...
www.ncbi.nlm.nih.gov
When SARS-CoV-2 invades cells, you get calcium pathways up, bradykinin up, angiotensin II up, and NRF2 down. This sets off the ROS cascade by promoting catastrophic superoxide release. Superoxide reacts with nitric oxide to form peroxynitrite (this is provably the case, because the biomarkers of elevated peroxynitrite, elevated nitrotyrosine, are present). Peroxynitrite actually "uncouples" endothelial nitric oxide synthase, the enzyme that makes nitric oxide, causing it to spew more superoxide instead. Neutrophils come along and start dumping extracellular superoxide dismutase and myeloperoxidase. Superoxide dismutase makes hydrogen peroxide from superoxide, and myeloperoxidase makes hypochlorous acid from hydrogen peroxide and chloride ions. Hypochlorous acid starts stripping iron from heme. Iron, superoxide, and hydrogen peroxide react in the Haber-Weiss and Fenton reactions to make deadly hydroxyl radicals that damage the linings of small capillaries in the lungs, causing edema, blood clots, and pneumonia.
All of this is preventable with strong antioxidants, particularly raising selenium and glutathione levels so that glutathione peroxidase has ample substrates, or raising vitamin D levels to pump calcium back out of cells and prevent the activation of NADPH oxidase, or taking curcumin (yes, turmeric pills) to activate NRF2 and improve endogenous antioxidant capacity. This actually ends up being antiviral against SARS-CoV-2, ultimately, because anything that lowers your superoxide levels will raise your nitric oxide levels, and nitric oxide is highly antiviral against SARS coronaviruses because it directly inhibits the Spike protein. You can also directly raise nitric oxide levels by eating foods high in dietary nitrate, like beets.
The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has spread worldwide and has seriously threatened public health by causing significant morbidity and mortality. Patients with coronavirus disease (COVID-19) with preexisting endothelial ...
www.ncbi.nlm.nih.gov
The science on this is pretty rock-solid at this point.
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